Patrinia scabiosifolia. P. villosa. Sonchus arvensis. 败 酱 草  Bài jiàng cǎo- "Defeat paste herb"    Patrinia    Family: Valerianaceae  
PART USED: Whole plant
Nature: Cool     FLAVOR: Bitter, Acrid, Pungent, harsh    CHANNEL: Stomach, Large Intestine, Liver
FUNCTIONS
GROUP: Clearing Internal Heat- Neutralizing Toxins
1. Clears Heat, relieves toxicity.[5] Relieve abscess. Promote pus drainage.[2]
2. Remove Blood stasis and alleviate pain.[3,5] Breaks up clots and stimulates circulation.[1] Eliminates swelling.[1,2] Transform/Expel coagulation.[2]
3. Clear Damp Heat.[2]
4. Tranquilizing.
INDICATIONS
1. Acute appendicitis.[1,2] Either internal Fire toxin disorders, such as intestinal abscess, or Fire toxin surfac sore and swellings.[5] Dysentery and enteritis.[1,2] Acupuncture point also useful- Lan wei- 2 cun below St 36.
2. Pain and obstruction associated with Heat induced Blood stasis, especially in the abdomen and chest.[5] Postpartum abdominal pain due to blood stasis.[5] Post partum abdominal cramps.[1] Menorrhagia. Abdominal pain due to blood stagnation.[2] Dispersing food, Qi or Blood, stasis due to Heat.[4]
Post operative pain.[5]
3. Boils and abscesses of unknown origin.[1] Carbuncles.[2] Tinea. Skin infection. Swelling.[2]
4. Hepatitis. Conjunctivitis.[1]
5. Pulmonary abscess.
6. Vexation, insomnia, insanity.[3]
CONTRAINDICATIONS: Overuse can cause fainting, nausea, decrease white blood cells.[4,5] Spleen and Stomach deficiency.[5]
COMBINATIONS
COMPARISONS
P. vilosa- pancreatitis, tonsillitis, influenza. P. scabiosifolia- also used for snake bite.[3]
PREPARATIONS: Decoction- Whole plant 9-30 g up to 30-90 g for single dose.[3] Decoction- Roots or whole plant 15-30 g.[1] Fresh tender leaves, crushed, may be used for external application over affected parts.[1,5] Whole plant 9-15 g.[2,5] Good quality has long roots, plentiful green leaves, and an intense fragrance.
  
ORIGIN: Grown in Central and Southern China, especially Sichuan, Jiangxi, Fujian.
References
Inner Path can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.
Constituents
Research
Decreases the white blood cell count.[4]
References
[1] Translation notes from Gary Seiford and Hocu Huhn- NSW College of Natural Therapies. Sydney Australia (1982).

Inhibitory effect of Patrinia scabiosifolia Link on the development of atopic dermatitis-like lesions in human keratinocytes and NC/Nga mice.
Cha KJ, Im MA, Gu A, Kim DH, Lee D, Lee JS, Lee JS, Kim IS.
Abstract
ETHNOPHARMACOLOGICAL RELEVANCE:
Atopic dermatitis (AD) is a chronic pruritic and inflammatory disease occurring in skin. Patrinia scabiosifolia Link (PS), a member of the Patrinia genus (Caprifoliaceae family), has traditionally been used in folk medicines to treat various inflammatory diseases such as acute appendicitis, ulcerative colitis, and pelvic inflammation in Korea and other parts of East Asia.
AIM OF THE STUDY:
This study investigated the anti-inflammatory effects of PS on AD in vitro and in vivo.
MATERIALS AND METHODS:
Whole PS plants were dried, powdered, and then underwent extraction with DMSO. Both ELISA and western blotting were performed to evaluate cytokine concentration and the expression and activation of filaggrin and signaling proteins. Five-week-old female NC/Nga mice were used as an AD-like mouse model by treating them with 2,4-dinitrochlorobenzene (DNCB).
RESULTS:
In human keratinocytic HaCaT cells, PS extract inhibited the production of IL-8, and TARC, which had been increased by TNF-α and IFN-γ. The TNF-α and IFN-γ suppressed filaggrin expression was associated with phosphorylation of JNK1 and JNK2, and NF-κB translocation. PS recovered the inhibition of filaggrin expression induced by TNF-α and IFN-γ by blocking the activation of JNK1/2, and NF-κB by the IFN-γ and TNF-α treatment. The in vivo experiment results showed that, compared to DNCB treatment PS administration reduced thickening of the epidermis and infiltration of inflammatory cells into the dermis. Moreover, the decrease of filaggrin expression due to DNCB treatment was recovered by PS administration. The serum IgE level was decreased by PS treatment. Additionally, secretions of IL-4, IL-5, IL-13, and eotaxin in splenocytes were lower in the PS-treated group than in the DNCB group.
CONCLUSION:
PS may attenuate the development of AD-like lesions by increasing filaggrin expression and lowering IgE and inflammatory cytokine levels. These results indicate the potential for development of a PS-based drug treatment for AD.
PMID: 28347830 DOI: 10.1016/j.jep.2017.03.045
J Ethnopharmacol. 2017 Jul 12;206:135-143. doi: 10.1016/j.jep.2017.03.045. Epub 2017 Mar 25. ncbi.nlm.nih.gov