Eucalyptus camaldulensis. Eucalyptus rostrata- River red gum and many other species of Eucalyptus
E. terminalis- Mountain bloodwood Family: Myrtaceae 
PART USED: Dried sap of the tree- Eucalyptus kino- The word “kino” is of West African origin.
DESCRIPTION: Dark reddish brown irregularly shaped pieces of gum.
TASTE: Astringent- adheres to the teeth and colors the saliva red.
ACTIONS
1. Astringent.[1,4,5]
2. Tonic.[1]
INDICATIONS
1. Diarrhea.[1,4,5] Inflamed mucous membranes and for diarrhea.[1] The Australian Aborigines, as well as early settlers, drank a solution of the red kino for diarrhea, as well as for chest and heart pains (perhaps due to indigestion).[2,3]
2. Sore throats- as a gargle.[1]
PREPARATIONS: 0.3-1.2 g mixed with water and drunk.[1,6]
Tincture of kino BPC1949 2-4 ml.[1]
Lozenge of Eucalyptus kino BPC1949.

PART USED: Leaves and twigs
INDICATIONS
1. Colds and fevers- an infusion of leaves and twigs has been used externally to bath the head.[2]
HABITAT: River banks, alluvial flats and damp, low-lying areas subject to flooding.
ORIGIN: E. rostrata and E. camadulensis originate from Australia, Madras and Sri Lanka.
DESCRIPTION
References
Inner Path can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.
Similar plants
Constituents
Research

Antibacterial Properties of Flavonoids from Kino of the Eucalypt Tree, Corymbia torelliana
Motahareh Nobakht, Stephen J. Trueman,* Helen M. Wallace, Peter R. Brooks, Klrissa J. Streeter, and Mohammad Katouli
Abstract
Traditional medicine and ecological cues can both help to reveal bioactive natural compounds. Indigenous Australians have long used kino from trunks of the eucalypt tree, Corymbia citriodora, in traditional medicine. A closely related eucalypt, C. torelliana, produces a fruit resin with antimicrobial properties that is highly attractive to stingless bees. We tested the antimicrobial activity of extracts from kino of C. citriodora, C. torelliana × C. citriodora, and C. torelliana against three Gram-negative and two Gram-positive bacteria and the unicellular fungus, Candida albicans. All extracts were active against all microbes, with the highest activity observed against P. aeruginosa. We tested the activity of seven flavonoids from the kino of C. torelliana against P. aeruginosa and S. aureus. All flavonoids were active against P. aeruginosa, and one compound, (+)-(2S)-4',5,7-trihydroxy-6-methylflavanone, was active against S. aureus. Another compound, 4',5,7-trihydroxy-6,8-dimethylflavanone, greatly increased biofilm formation by both P. aeruginosa and S. aureus. The presence or absence of methyl groups at positions 6 and 8 in the flavonoid A ring determined their anti-Staphylococcus and biofilm-stimulating activity. One of the most abundant and active compounds, 3,4',5,7-tetrahydroxyflavanone, was tested further against P. aeruginosa and was found to be bacteriostatic at its minimum inhibitory concentration of 200 µg/mL. This flavanonol reduced adhesion of P. aeruginosa cells while inducing no cytotoxic effects in Vero cells. This study demonstrated the antimicrobial properties of flavonoids in eucalypt kino and highlighted that traditional medicinal knowledge and ecological cues can reveal valuable natural compounds.
Plants (Basel). 2017 Sep; 6(3): 39.
Published online 2017 Sep 14. doi: 10.3390/plants6030039
PMCID: PMC5620595 PMID: 28906457 ncbi.nlm.nih.gov

Development of a new model for the induction of chronic kidney disease via intraperitoneal adenine administration, and the effect of treatment with gum acacia thereon
Mohammed Al Za’abi, Mahfouda Al Busaidi, Javid Yasin, Nicole Schupp, Abderrahim Nemmar, and Badreldin H Ali
Abstract
Oral adenine (0.75% w/w in feed), is an established model for human chronic kidney disease (CKD). Gum acacia (GA) has been shown to be a nephroprotective agent in this model. Here we aimed at developing a new adenine-induced CKD model in rats via a systemic route (intraperitoneal, i.p.) and to test it with GA to obviate the possibility of a physical interaction between GA and adenine in the gut. Adenine was injected i.p. (50 or 100 mg/Kg for four weeks), and GA was given concomitantly in drinking water at a concentration of 15%, w/v. Several plasma and urinary biomarkers of oxidative stress were measured and the renal damage was assessed histopathologically. Adenine, at the two given i.p. doses, significantly reduced body weight, and increased relative kidney weight, water intake and urine output. It dose-dependently increased plasma and urinary inflammatory and oxidative stress biomarkers, and caused morphological and histological damage resembling that which has been reported with oral adenine. Concomitant treatment with GA significantly mitigated almost all the above measured indices. Administration of adenine i.p. induced CKD signs very similar to those induced by oral adenine. Therefore, this new model is quicker, more practical and accurate than the original (oral) model. GA ameliorates the CKD effects caused by adenine given i.p. suggesting that the antioxidant and anti-inflammatory properties possessed by oral GA are the main mechanism for its salutary action in adenine-induced CKD, an action that is independent of its possible interaction with adenine in the gut.
Am J Transl Res. 2015; 7(1): 28–38.
Published online 2015 Jan 15.
PMCID: PMC4346521 PMID: 25755826 ncbi.nlm.nih.gov